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2015). These scientific studies, taken together, recommend that, Though a direct activation of GPR55 or PPAR?? takes place, PEA can make its anti?inflammatory action from the intestine also via indirect activation of CB1 and CB2 receptors, in all probability as a consequence of the flexibility of the compound to potentiate the motion of endocannabi
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Taken alongside one another, PEA and its congeners could depict an impressive therapeutic system inside the management of combined neuropathies.The anti?inflammatory outcomes of PEA appear to be mainly linked to its ability to modulate mast cell activation and degranulation, and this action is often called the ALIA (autacoid regional inflammation a
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